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A small molecule interacts with VDAC2 to block mouse BAK-driven apoptosis
Mice Haploinsufficient for Ets1 and Fli1 Display Middle Ear Abnormalities and Model Aspects of Jacobsen Syndrome
Caspase-9 mediates the apoptotic death of megakaryocytes and platelets, but is dispensable for their generation and function
Expansion of the neonatal platelet mass is achieved via an extension of platelet lifespan
Mcl-1 and Bcl-xL coordinately regulate megakaryocyte survival
Physiological restraint of Bak by Bcl-xL is essential for cell survival
Megakaryocytes possess a functional intrinsic apoptosis pathway that must be restrained to survive and produce platelets
Platelet production proceeds independently of the intrinsic and extrinsic apoptosis pathways
