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Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.
The Ripoptosome, a signaling platform that assembles in response to genotoxic stress and loss of IAPs
IAPs are functionally non-equivalent and regulate effector caspases through distinct mechanisms
IAP-antagonists exhibit non-redundant modes of action through differential DIAP1 binding
Jafrac2 is an IAP antagonist that promotes cell death by liberating Dronc from DIAP1
The DIAP1 RING finger mediates ubiquitination of Dronc and is indispensable for regulating apoptosis
