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Common genetic variants, acting additively, are a major source of risk for autism
On the use of general control samples for genome-wide association studies: genetic matching highlights causal variants
Mapping autism risk loci using genetic linkage and chromosomal rearrangements
Individual common variants exert weak effects on the risk for autism spectrum disorders
Convergence of genes and cellular pathways dysregulated in autism spectrum disorders
Autism genome-wide copy number variation reveals ubiquitin and neuronal genes
A genome-wide scan for common alleles affecting risk for autism
